In their paper, they identify soluble factors that are induced in the secretome of NIH-CXCL14 fibroblasts compared to negative control ones, and propose that some of the molecules specifically enriched in those cells, among which many pro-angiogenic factors, inducers of EMT and of extracellular matrix remodeling, and several other chemokines, may be the direct effectors of CXCL14 action on tumor cells. The gene discussed is CXCL14; the disease is neoplasm.