Upon infection of BmN cells with BmNPV, the inductionof apoptosis increased, although HN1L overexpression could inhibit DNA fragmentation,suggesting that the HN1L protein could inhibit cell apoptosis induced by viral invasion.In addition, Western blotting indicated that the HN1L protein inhibited the activation ofcaspase-9 zymogen and the expression of Bax protein, although it promoted Bcl-2expression. Here, BAX is linked to infection.