After hepatic reperfusion (T2), the non-ARDS group had a significant increase in IL-10 (173.2 ± 155.8 vs. 44.3 ± 126.6 pg.mL−1 at T2 and T1, respectively; p = 0.0002; Figure 1(b)) and a trend of increased HMGB1 (70.5 ± 112.8 vs. 33.2 ± 58.8 ng.mL−1 at T2 and T1, respectively; p = 0.0884; Figure 2(a)) compared with the baseline (T1) values. This evidence concerns the gene HMGB1 and acute respiratory distress syndrome.