Others showed that the knockdown of FABP4 increased survival in a HoxA9/Meis1-induced AML mouse model, and in vitro cocultures of adipocytes differentiated from BM MSC together with AML blasts showed activation of lipolysis and transfer of FA from adipocytes to AML blasts with participation of FABP4 [139] (Figure 6). This evidence concerns the gene HOXA9 and acute myeloid leukemia.