Previously, Huang W et al. reported Bevacizumab induced EMT phenotype in glioblastoma cell line U87MG and the overexpression of BATF2 (basic leucine zipper ATF-like transcription factor 2), a multi-target transcriptional repressor, significantly inhibited Bevacizumab-induced EMT with suppression of Wnt/β-catenin signaling (43). The gene discussed is BATF2; the disease is glioblastoma.