BRCA2 and neoplasm: SCYP3 produced outside the meiotic context has been shown to disrupt the activity of the tumour‐suppressing recombination regulator BRCA2 and can modulate the strand invasion capabilities of both the RAD51 and DMC1 (meiosis‐specific) recombinases that drive homologous recombination (Hosoya et al., 2011; Kobayashi et al., 2017) (Fig. 3).