Similar studies have determined the effects of the endotoxininduced endophthalmitis and the experimental diabetic retinopathy in the blood-retinal barrier function [131,132], as well as ischemic-induced injuries [133] and their hyperpermeability after the sustained elevation of the vascular endothelial growth factor (VEGF) in the vitreous cavity [134]. This evidence concerns the gene VEGFA and diabetic retinopathy.