While the level of gene expression was stable, CIDEB was the most highly expressed among the 3 members of the CIDE family in hepatocytes (2000x more than CIDEA and 400x more than FSP27/CIDEC in mice and humans) and could be an important player in the development of hepatic steatosis since it regulates the formation of triacylglycerol-enriched VLDL particles36. The gene discussed is CIDEB; the disease is Hepatic steatosis.