In addition, the data in Fig. 6d showed that, compared with UUO WT mice, FABP4 knockdown attenuated lipid metabolism disorders via inhibiting PPARγ signal by affecting the protein levels of ACADL, ACADM, CPT1, ACOX1, SCP-2, and EHHADH in UUO FABP4 KO mice (Details of fold changes and significances of these proteins in Western blot assay are shown in Supplemental Fig. S4). The gene discussed is FABP4; the disease is Disorder of lipid metabolism.