Nonetheless, it has been demonstrated that ectopic expression of CXCR2 in T-cells from normal donors or tumor-associated lymphocytes from ovarian cancer patients can increase their migration towards recombinant IL-8 and autologous ascites that contains CXCL1 and CXCL8 (IL-8) [30,54]. The gene discussed is CXCR2; the disease is neoplasm.