LPC family members, especially LPC (18:0), were proved to be a critical factor underlying cardiovascular diseases [29], pneumonia [30], Alzheimer’s disease [31], hypobaric hypoxia [32], hepatitis B [33], and several pathological conditions that are associated with elevated LPC levels in the circulation [34,35,36]. This evidence concerns the gene PCSK7 and early-onset autosomal dominant Alzheimer disease.