This discrepancy could be explained by the differences in methodology adopted such as the type of animal model, muscle used (e.g., in Veskoukis et al. gastrocnemius, which has a different myosin composition than rabbit psoas), lab protocols, and the renal dysfunction per se having possibly an overarching systemic effect that could mask muscle's contribution to blood levels of redox indices. The gene discussed is MYH14; the disease is Abnormal renal physiology.