In contrast, we observed significantly enhanced expression of Cxcl1 (KC), Il6, and Il17, but not of Cxcl2 (MIP-2), Ifng, or Il10, in the huLangerin group as opposed to WT controls after infection with the S. aureus ΔtarM mutant (Fig. 4C). The gene discussed is IFNG; the disease is infection.