For instance, sulforaphane has been reported to induce apoptosis and cell cycle arrest of human colon cancer cells by increasing cyclins A and B1 expression [2]; the administration of sulforaphane shows antitumor formation [9]; sulforaphane inhibits the Aβ1–42-induced excessive secretion of interleukin-1β (IL-1β) in human microglia-like cells via activation of nuclear factor erythroid 2-related factor 2 (Nrf2), a bZIP transcription factor and an inducer of phase 2 genes [10]. Here, NFE2L2 is linked to colonic neoplasm.