It is very plausible that this effect was associated with the higher secretion levels of several agents known to support ovarian cancer cell progression by these cells, such as CCL2, CXCL8, CXCL12, ICAM-1, IL-6, HGF, PAI-1, uPA, TGF-β1, and VEGF [2]. This evidence concerns the gene CXCL12 and ovarian carcinoma.