An in vivo xenograft model showed that SBF2-AS1 overexpression promoted lung cancer growth; however, when the binding sites of miR-338-3p and miR-362-3p were both mutated (SBF2-AS1-MUT), SBF2-AS1 failed to promote growth of lung cancer (Figure 6A). This evidence concerns the gene SBF2 and lung cancer.