We also ran this analysis in parallel for data from hematopoietic progenitors of patients with myelodysplastic syndrome (MDS), a disorder well-known for significant impairments in terminal erythropoiesis, either with or without somatic mutations in the related splicing factor SF3B1 (Obeng et al., 2016). This evidence concerns the gene SF3B1 and myelodysplastic syndrome.