APLN and lung adenocarcinoma: Our data revealed that miR‐195 significantly inhibited the firefly luciferase activity of the vector with the wild‐type 3′‐UTR of apelin; however, there was no significant effect on the vector with the mutated 3′‐UTR of apelin in A549 and GLC‐82 lung adenocarcinoma cells (Fig 3b).