In addition to the Wnt/ β-catenin pathway, the inactivating mutations TP53 have been associated with defects in the ability of tumor cells to produce key chemokines required for BAFT3 DC recruitment [38]; p53 loss of function and lack of T-cell infiltration have been found in basal-like ER-negative breast cancers, but not in ER-positive breast cancer [39]. Here, TP53 is linked to neoplasm.