Although cell surface receptors that lead to hRSV cell infection have been identified, such as cellular heparan sulfate glycosaminoglycans that act as attachment factors for the hRSV G glycoprotein (75, 76), as well as nucleolin (37) and ICAM1 (77) as ligands for the F fusion protein, the exact mechanism by which hRSV enters DCs has not been corroborated and could eventually be different compared to that observed in other cells, such as epithelial cells (78). This evidence concerns the gene NUCLEOLIN and infection.