FN1 and infective endocarditis: Complementation studies involving TlyC turned a nonhemolytic bacteria into an hemolytic one [65] and FbpA is considered an important factor in infective endocarditis, with the S. suis FbpA featuring both the N-terminal fibrinogen binding domain and the C-terminal DUF814 domain found in Enterococcus faecalis homologs shown to bind to immobilized fibronectin [66].