LGALS3 and acute myocardial infarction: This finding is in line with the knowledge that if Galectin 3 binding protein has a role in the inflammatory process which leads to progression and destabilization of atherosclerotic plaques [13], it should be highly expressed in patients who suffered acute myocardial infarction, which in most cases is due to a thrombotic occlusion of a coronary artery, following a plaque rupture [28].