MICAL‐L2, a member of MICAL family, is present abundantly in ovarian cancer tissues.7 Silencing of MICAL‐L2 could suppress malignancy of ovarian cancer cells by inhibiting canonical Wnt/β‐catenin signalling and inducing mesenchymal‐epithelial transition.7 Furthermore, MICAL‐L2 was also identified preferentially providing ‘law and order' in collective cell migration.8 Although the results above suggested that MICAL‐L2 may be involved in cancer cell invasion and metastasis while our knowledge on functions of MICAL‐L2 is limited. The gene discussed is MICAL1; the disease is cancer.