For example, increased liver iron levels are detected in approximately 30% of unselected patients with NAFLD,9 whereas an iron‐deficient diet improves insulin sensitivity in patients with NAFLD.10 Animal experiments revealed that a marginal iron deficiency diet enhances de novo lipogenesis in rats fed with a sucrose diet.11 Moreover, plasma selenium levels are increased with NAFLD risk in humans.12 Indeed, selenium exposure increases liver TG concentrations. The gene discussed is INS; the disease is metabolic dysfunction-associated steatotic liver disease.