Supporting this result, a neuro‐image study recently reported that carriers of FTO‐obesity risk variants have a reduced longitudinal functioning in the medial prefrontal cortex and this reduction correlated with higher rates of impulsivity, behavioral disinhibition, and risky decision‐making, which are common symptoms in BD (Chuang et al., 2015). Here, FTO is linked to obesity due to melanocortin 4 receptor deficiency.