The expression of CD64 on monocytes has been examined in SLE patients and was found to be increased in active systemic inflammation and be related to the development of nephritis.22 These results were concordant with the study done by Liu et al in HT patients.15 In addition, we also found cell percentages expressed CD16 were also similar among three groups, which was inconsistent with those in HT study. This evidence concerns the gene FCGR1A and nephritis.