FCGR2B and hematocrit: A study about Hashimoto's thyroiditis recently implied that increasing expression of FcγRIII and decreasing expression of FcγRIIB may be involved in the pathogenesis of HT.15 The expression of FcγRII from GD patients was reported by Estienne et al, and the authors suggested that FcγRII expressed by thyrocytes might involve in eliminating IgG.16