PRKCD and Parkinson disease: PKCδ‐induced mitochondrial dysfunction is also associated with dopaminergic neurotoxicity in preclinical PD models.143, 145 Although the outcomes of mitochondrial PKCδ signaling affect apoptosis, respiration, membrane potential, ROS production, antioxidant capacity, and organelle dynamics,146, 147, 148 only Drp‐1 has been shown to interact with PKCδ and contribute to organelle fragmentation.149 Further investigation is needed to elaborate on PKCδ functions at mitochondria.