Since EHV1 has a prominent ability to infect immune cells and more efficiently infects (IL-2) activated T-lymphocytes (van der Meulen et al., 2000; Poelaert et al., 2019), we hypothesized that SCFA might reduce EHV1 infection of mononuclear cells by interfering with immune cell proliferation or by changing cellular phenotypes, and thus impeding the cell-associated viremia. This evidence concerns the gene IL2 and infection.