Inhibiting cardiomyocyte necrosis by suppressing receptor interacting protein kinase 1/3 (RIPK1/3) RNA synthesis and protein posttranslation decreases myocardial infarct size in an ischemia/reperfusion (I/R) injury animal model (Wang K. et al., 2016). This evidence concerns the gene RIPK1 and myocardial infarction.