For examples, MIR376B plays an oncogenic role by directly reducing BECN1 and ATG4C protein translation, and attenuates rapamycin‐induced autophagy in Huh‐7 and MCF‐7 cells,9 whereas MIR30A negatively regulates BECN1 expression and decreases autophagy activity in rapamycin‐treated T98G cells.10 Herein, we found that MIR190A promoted BC invasion and autophagy via stabilizing ATG7 mRNA by directly binding to its 3′‐untranslated region (UTR). Here, MIR190A is linked to breast cancer.