In addition, it is highly significant to note that xenograft tumor formation and growth, as demonstrated in tumor size and tumor weight, also showed that ectopic expression of MIR190A promoted tumor formation and growth, while knockdown of ATG7 in UMUC3(MIR190A) cells completely abolished the promotive effect of MIR190A on xenograft tumor growth (Figure 8H–J). The gene discussed is ATG7; the disease is neoplasm.