Considering the previous reports regarding the role of nELAVL Hu-proteins in neuronal excitability by binding to the mRNAs encoding proteins from the glutamate synthesis pathway [15], or encoding Kv1.1 voltage-gated potassium channels [16], we might suppose that nELAVL Hu-proteins might also stabilize / regulate mRNA encoding other proteins (i.e., ion channels) involved in DRG neuronal excitability and being important players in the algesic profile and diabetes. The gene discussed is KCNA1; the disease is diabetes mellitus.