The FGFR3-BAIAP2L1 fusion promotes ligand-independent phosphorylation of FGFR3-BAIAP2L1 in Rat-2_F3-B cells and tumor formation in nude mice inoculated with Rat-2_F3-B cells [16] by activating ERK and STAT1 signaling [48], similar to FGFR2-CCDC6 [48]. The gene discussed is FGFR2; the disease is neoplasm.