Moreover, we found by microarray analysis of the dura of MC-deficient WBB6F1-KitW/W-v mice, the corresponding WT (WBB6F1-Kit+/+) mice, and WBB6F1-KitW/W-v mice which had been engrafted in the meninges with WT BMCMCs (unpublished data), that the meninges are a site of inflammation-related activity after stroke and that the meningeal inflammatory gene response to stroke is modulated, at least in part, by MCs (Figure 3A). This evidence concerns the gene KIT and stroke disorder.