Moreover, we found by microarray analysis of the dura of MC-deficient WBB6F1-KitW/W-v mice, the corresponding WT (WBB6F1-Kit+/+) mice, and WBB6F1-KitW/W-v mice which had been engrafted in the meninges with WT BMCMCs (unpublished data), that the meninges are a site of inflammation-related activity after stroke and that the meningeal inflammatory gene response to stroke is modulated, at least in part, by MCs (Figure 3A). The gene discussed is KIT; the disease is Stroke.