ITGB8 and mastocytosis: This delay was not associated with differences in other proposed mechanisms involved in helminth expulsion, with no significant difference in crypt/villus architecture (S2D Fig), goblet cell hyperplasia [42] (S2E Fig), mastocytosis [18–20] and associated MMCP-1 production [21,22] (S2F and S2G Fig) or RELMβ expression [43] (S2H Fig) between wild-type and Itgb8 (CD11c-Cre) mice.