Hedgehog signaling was activated in NSCLCs, and several Hedgehog components, including PTCH1, SMO, and GLI1, were correlated with the increased expression of FOXM1, indicating that overexpression of FOXM1 may be a new mechanism by which SHH signaling induces cell proliferation in NSCLC; however, these results need to be investigated further [41]. Here, FOXM1 is linked to non-small cell lung carcinoma.