Interestingly, Sun L. et al. identified that inhibition of FOXM1 by RCM-1 reduced IL-13 and STAT6 signaling, prevented the expression of the STAT6 target genes Spdef and Foxa3 in goblet cells, prevented goblet cell metaplasia, decreased airway resistance, and reduced lung inflammation in mice in response to HDM and recombinant IL-13, providing a new potential therapeutic for asthma [69]. The gene discussed is FOXM1; the disease is asthma.