FOXM1 and Sepsis: However, the rapid resolution of lung inflammation induced by BMPCs also requires endothelial expression of FOXM1, which is consistent with previous research showing that FOXM1 is markedly induced in lung endothelial cells but during only the repair phase following sepsis-induced lung injury, while FOXM1 expression is silenced in pulmonary vascular endothelial cells in adult lungs [78, 83].