GAL and sensory ataxia: Meanwhile, abundant of GD1b are also found in the large neurons of human dorsal root ganglia, sympathetic ganglia, as well as the paranodal myelin of motor and peripheral sensory nerves.[12] The loss of GD1b may cause the abnormality of motor and sensation, and sensory ataxia.[13–15] Interestingly, some autoantibodies show affinity for the terminal Gal (β1–3) GalNAc residue, which is shared by GM1 and GD1b[16] (Table 2).