First, for K562 CML cells, INKA analysis pinpointed ABL1 as a prime candidate and inferred phosphorylation of its downstream signaling partners such as SRC‐family kinases and MAPK1/3 (Figs 2A and 3A, Appendix Fig S2). This evidence concerns the gene ABL1 and chronic myelogenous leukemia, BCR-ABL1 positive.