Then, the up‐regulation of CCND2 was closely correlated with poor prognosis of glioma patients and CCND2 knockdown suppresses the cellular proliferation, migration, invasion and EMT in glioma cells via miR‐185‐5p and FOXD2‐AS1 regulation.28 Another example, NEAT1 and CDK6 could promote the proliferation and metastasis of glioma cells and inhibit cell apoptosis, while the miR‐139‐5p suppresses the tumour effect on the biological functions of glioma cells.29 This evidence concerns the gene NEAT1 and central nervous system cancer.