Abnormal apoptoses, such as in apoptotic keratinocytes and colloid bodies, are often found in OLP patients,6, 8, 36 specifically in the basal layer.19, 37 González‐Moles et al38 reported that apoptosis was not consistently present in all OLP tissues, as evidenced by the differential expression of caspase 3 and Bcl2, further suggesting that pathological changes might be heterogeneous in different layers of OLP tissues. The gene discussed is BCL2; the disease is oral lichen planus.