For instance, the activation of mTOR‐dependent autophagy may delay neurodegenerative symptoms in fly and mouse models of Huntington's disease,35 and a mouse model of Alzheimer's disease.36 The present study found that with increased exposure time, the levels of p‐mTOR gradually decreased, which was negatively correlated with changes in p‐ULK1 and LC3II. This evidence concerns the gene ULK1 and juvenile Huntington disease.