A systematic review concluded that inflammation was the main precipitating factor for the occurrence of AF after cardiac surgery and introduced drugs with anti-inflammatory properties for the prevention of this arrhythmia.2 Elevations in inflammation markers such as C-reactive protein (CRP) and interleukin 6, concurrently with postoperative AF, confirm this theory.3, 4 Other suggested mechanisms are neurohormonal activation,5 increased sympathetic and parasympathetic activities,6 and oxidative stress.7 This evidence concerns the gene CRP and atrial fibrillation.