EGFR and glioblastoma: For example, inhibition of VEGFA/VEGFR2 expression suppresses GBM growth (Serrill et al., 2016); EGFR suppression hinders GBM growth (Shen et al., 2018); MYC activation in GBM drives glycolysis, thereby fueling pathogenesis of GBM (Tateishi et al., 2016); CD44 expression is overexpressed in GBM and the increased expression of CD44 promotes GBM aggressiveness including mediation of migration (Chetty et al., 2012; Mooney et al., 2016).