Deacetylated FoxO1 protects β‐cell function by limiting mitochondrial lipid utilization through decreasing fatty acid oxidation and enhancing insulin secretion in the context of diabetes (Kim‐Muller et al., 2016); which is in consist in the findings that the levels of acetylated FoxO1 are markedly increased in pancreatic tissues of diabetic, compared to healthy rats (Ding et al., 2014). The gene discussed is FOXO1; the disease is diabetes mellitus.