Because IL‐36α is induced not only by IMQ but also by β‐glucans from Candida albicans, fungal infection may also be involved in the development of dermatitis.80 It is likely that various innate immune receptors such as TLRs and CLRs expressed on the cell surface of LCs and DCs of the skin recognize PAMPs of bacteria and fungi, as well as alarmins derived from dying cells, and induce cytokines and chemokines including IL‐36α, IL‐23, IL‐1β, and CCL20. Here, IL36A is linked to fungal infectious disease.