As panobinostat mitigated IRF1 protein levels in MM cells in the presence of IFN-γ over time (Figure 3D), and because PD-L1 is a target gene of STAT1 as well as IRF1 [19–22], we further clarified the role of STAT1 in the upregulation of PD-L1 in MM cells by panobinostat and IFN-γ in combination. The gene discussed is IFNG; the disease is Miyoshi myopathy.