To further determine the role of G0S2 in glioma response to IR, we stably overexpressed G0S2 in two glioma cell lines with low levels of endogenous G0S2 expression (Fig. 3a), LN229 and U87, and then treated U87/G0S2, U87/Control, LN229/G0S2, LN229/Control, U251/G0S2, and U251/Control with fractionated radiation. The gene discussed is G0S2; the disease is central nervous system cancer.