NT5E and myocardial infarction: In addition, the local treatment with MSCs promoted the CD73 expression on host monocytes in a swine model of myocardial infarction, and the upregulation of CD73 on MSC‐conditioned monocytes was an effective mechanism to amplify the long‐lasting immunomodulatory and healing effects of MSCs delivery.43 We consistently showed that the expression of CD73 on NK cells co‐cultured in contact with DPSCs or BMMSCs had significant differences compared with transwell co‐culture system.