FGFR3 and neoplasm: We have shown that ETV5 is a downstream target of mutant FGFR3 through the MAPK/ERK signalling pathway, is involved in the cross-talk between FGFR3 signalling and the Hippo pathway, upregulates the expression of genes involved in EMT in cell derived from invasive tumours, and mediates proliferation and anchorage-independent growth of bladder cancer cells.