DUSP14 and cardiac hypertrophy: Cardiac‐specific overexpression of DUSP8 causes spontaneous eccentric remodelling and ventricular dilation with heart failure.39 DUSP14 prevents cardiac hypertrophy and dysfunction induced by aortic banding by inactivating the TAK1/p38MAPK/JNK1/2 signalling pathway.40 In addition, heat shock protein 90 regulates cardiac ventricular hypertrophy through the activation of MAPK pathway.41 In brief, phosphorylation modifications play important roles in the regulation of cardiac hypertrophy and may prove to be promising targets for therapeutic development.