A recurrent loss of ADAMTS9 (3p14.1) was found in our cohort, consistent with previous fluorescence in situ hybridization findings.27 Arm‐level losses of FHIT (3p14.1‐p14.3) and other NPC‐related tumor suppressor genes28 were discovered in six NPC patients (6/82, 7.3%) and are concordant with previous findings.10 Recurrent PTPRG and HepaCAM deletions were newly detected in NPC patients (3p14, 7.3%, 6/82 and 11q24.1‐q24.2, 11.0%, 9/82, respectively). Here, ADAMTS9 is linked to nasopharyngeal carcinoma.